TNF-α/STAT1/CXCL10 mutual inflammatory axis that contributes to the pathogenesis of experimental models of multiple sclerosis: A promising signaling pathway for targeted therapies
Cytokine, vol. 168, pp. 156235
Authors 6
-
Affiliation as printed
Department of Biology, Faculty of Sciences, University of Zabol, Zabol, Iran
-
North Khorasan University of Medical Sciences
Affiliation as printed
Department of Medical Biotechnology and Molecular Science, North Khorasan University of Medical Science, Bojnurd, Iran
-
Affiliation as printed
Department of Biology, Faculty of Sciences, University of Zabol, Zabol, Iran
-
Universitätsklinikum Aachen · RWTH Aachen University
Affiliation as printed
Institute of Neuroanatomy, RWTH University Hospital Aachen, 52074 Aachen, Germany
-
Zabol University · Universitätsklinikum Aachen · RWTH Aachen University
Affiliation as printed
Department of Biology, Faculty of Sciences, University of Zabol, Zabol, Iran; Institute of Neuroanatomy, RWTH University Hospital Aachen, 52074 Aachen, Germany. Electronic address: nsanadgol@ukaachen.de
Department of Biology, Faculty of Sciences, University of Zabol, Zabol, Iran
Institute of Neuroanatomy, RWTH University Hospital Aachen, 52074 Aachen, Germany
-
Affiliation as printed
Institute of Anatomy, Department of Biomedicine, University of Basel, 4001 Basel, Switzerland
Cited by 17 stored of 18
17 results
No patents citing this paper on Lens.org (checked 2026-10-06).